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Iron-deficiency anemia
sufficient amounts of iron or by iron supplementation. Foods high in iron include meat, nuts, spinach, and foods made with iron-fortified flour. Treatment

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Iron-deficiency anemiaSynonymsIron-deficiency anaemiaRed blood cellsSpecialtyHematologySymptomsFeeling tired, weakness, shortness of breath, confusion, pallor[1]ComplicationsHeart failure, arrhythmias, frequent infections[2]CausesIron deficiency[3]Diagnostic methodBlood tests[4]TreatmentDietary changes, medications, surgery[3]MedicationIron supplements, vitamin C, blood transfusions[5]Frequency1.48 billion (2015)[6]Deaths54,200 (2015)[7]

Iron-deficiency anemia is anemia caused by a lack of iron.[3] Anemia is defined as a decrease in the number of red blood cells or the amount of hemoglobin in the blood.[3] When onset is slow, symptoms are often vague such as feeling tired, weak, short of breath, or having decreased ability to exercise.[1] Anemia that comes on quickly often has more severe symptoms, including: confusion, feeling like one is going to pass out or increased thirst.[1] Anemia is typically significant before a person becomes noticeably pale.[1] Children with iron deficiency anemia may have problems with growth and development .[3] There may be additional symptoms depending on the underlying cause.[1]

Iron-deficiency anemia is caused by blood loss, insufficient dietary intake, or poor absorption of iron from food.[3] Sources of blood loss can include heavy periods, childbirth, uterine fibroids, stomach ulcers, colon cancer, and urinary tract bleeding.[8] Poor absorption of iron from food may occur as a result of Crohn's disease or a gastric bypass.[8] In the developing world, parasitic worms, malaria, and HIV/AIDS increase the risk of iron deficiency anemia.[9] Diagnosis is confirmed by blood tests.[4]

Iron deficiency anemia can be prevented by eating a diet containing sufficient amounts of iron or by iron supplementation.[10] Foods high in iron include meat, nuts, spinach, and foods made with iron-fortified flour.[11] Treatment may include dietary changes and dealing with underlying causes, for example medical treatment for parasites or surgery for ulcers.[3] Iron supplements and vitamin C may be recommended.[5] Severe cases may be treated with blood transfusions or iron injections.[3]

Iron-deficiency anemia affected about 1.48 billion people in 2015.[6] A lack of dietary iron is estimated to cause approximately half of all anemia cases globally.[12] Women and young children are most commonly affected.[3] In 2015 anemia due to iron deficiency resulted in about 54,000 deaths – down from 213,000 deaths in 1990.[7][13]

.mw-parser-output .toclimit-2 .toclevel-1 ul,.mw-parser-output .toclimit-3 .toclevel-2 ul,.mw-parser-output .toclimit-4 .toclevel-3 ul,.mw-parser-output .toclimit-5 .toclevel-4 ul,.mw-parser-output .toclimit-6 .toclevel-5 ul,.mw-parser-output .toclimit-7 .toclevel-6 ul{display:none}Contents
  • 1 Signs and symptoms
    • 1.1 Child development
  • 2 Cause
    • 2.1 Parasitic disease
    • 2.2 Blood loss
      • 2.2.1 Menstrual bleeding
      • 2.2.2 Gastrointestinal bleeding
    • 2.3 Diet
    • 2.4 Iron malabsorption
    • 2.5 Pregnant women
    • 2.6 Children
    • 2.7 Blood donation
  • 3 Mechanism
  • 4 Diagnosis
    • 4.1 Blood tests
    • 4.2 Screening
  • 5 Treatment
  • 6 Epidemiology
  • 7 References
  • 8 External links
Signs and symptoms

Iron deficiency anemia may be present without a person experiencing symptoms.[14] If symptomatic, patients may present with the sign of pallor (reduced oxyhemoglobin in skin or mucous membranes), and the symptoms of fatigue, lightheadedness, decreased exercise tolerance, headache, and weakness.[14] None of these symptoms (or any of the others below) are sensitive or specific. The symptom most suggestive of iron deficiency anemia in children is pallor of mucous membranes (primarily the conjunctiva). Even so, a large study showed that pallor of the mucous membranes is only 28% sensitive and 87% specific (with high predictive value) in distinguishing children with anemia (defined as hemoglobin < 11.0 g/dl) and 49% sensitive and 79% specific in distinguishing severe anemia (hemoglobin < 7.0 g/dl).[15] Thus, this sign is reasonably predictive when present, but not helpful when absent, as only one-third to one-half of children who are anemic (depending on severity) will show pallor.

Iron deficiency anemia tends to develop slowly; therefore the body has time to adapt, and the disease often goes unrecognized for some time.[16] In severe cases, shortness of breath can occur.[17] Pica may also develop; of which consumption of ice, known as pagophagia, has been suggested to be the most specific for iron deficiency anemia.[16]

Other possible symptoms and signs of iron-deficiency anemia include:[3][16][17][18]

Koilonychia (spoon-shaped nails)
  • Irritability
  • Angina (chest pain)
  • Palpitations (feeling that the heart is skipping beats or fluttering)
  • Breathlessness
  • Tingling, numbness, or burning sensations
  • Glossitis (inflammation or infection of the tongue)
  • Angular cheilitis (inflammatory lesions at the mouth's corners)
  • Koilonychia (spoon-shaped nails) or nails that are brittle
  • Poor appetite
  • Dysphagia (difficulty swallowing) due to formation of esophageal webs (Plummer-Vinson syndrome)
  • Restless legs syndrome[19]
Child development

Iron-deficiency anemia is associated with poor neurological development, including decreased learning ability and altered motor functions.[20][21] This is because iron deficiency impacts the development of the cells of the brain called neurons. When the body is low on iron, the red blood cells get priority on iron and it is shifted away from the neurons of the brain. Exact causation has not been established, but there is a possible long-term impact from these neurological issues.[21]


A diagnosis of iron-deficiency anemia requires further investigation into its cause.[22] It can be caused by increased iron demand, increased iron loss, or decreased iron intake.[23] Increased iron demand often occurs during periods of growth, such as in children and pregnant women.[24] For example, during stages of rapid growth, babies and adolescents may outpace their dietary intake of iron which can result in deficiency in the absence of disease or a grossly abnormal diet.[23] Iron loss is typically from blood loss. [24] One example of blood loss is by chronic gastrointestinal blood loss, which could be linked to a possible malignancy.[22] In women of childbearing age, heavy menstrual periods can be a source of blood loss causing iron-deficiency anemia.[22] People who do not consume much iron in their diet, such as vegans or vegetarians, are also at increased risk of developing iron deficiency anemia.[14]

Parasitic disease

The leading cause of iron-deficiency anemia worldwide is a parasitic disease known as a helminthiasis caused by infestation with parasitic worms (helminths); specifically, hookworms. The hookworms most commonly responsible for causing iron-deficiency anemia include Ancylostoma duodenale, Ancylostoma ceylanicum, and Necator americanus.[22][25] The World Health Organization estimates that approximately two billion people are infected with soil-transmitted helminths worldwide.[26] Parasitic worms cause both inflammation and chronic blood loss by binding to a human's small-intestinal mucosa, and through their means of feeding and degradation, they can ultimately cause iron-deficiency anemia.[25][16]

Blood loss

Red blood cells contain iron, so blood loss also leads to a loss of iron. There are several causes of blood loss including menstrual bleeding, gastrointestinal bleeding, stomach ulcers, and bleeding disorders.[27] The bleeding may occur quickly or slowly. Slow, chronic blood loss within the body — such as from a peptic ulcer, angiodysplasia, inflammatory bowel disease, a colon polyp or gastrointestinal cancer (e.g., colon cancer)— can cause iron-deficiency anemia.

Menstrual bleeding

Menstrual bleeding is a common cause of iron deficiency anemia in women of child bearing age.[27] Women with menorrhagia (heavy menstrual periods) are at risk of iron-deficiency anemia because they are at higher-than-normal risk of losing a larger amount blood during menstruation than is replaced in their diet. Most women lose about 40 mL of blood per cycle. Iron is lost with the blood. Some birth control methods, such as pills and IUDs, may decrease the amount of blood, therefore iron lost during a menstrual cycle.[27]

Gastrointestinal bleeding

The most common cause of iron deficiency anemia in men and post-menopausal women is gastrointestinal bleeding.[27] There are many sources of gastrointestinal tract bleeding including the stomach, esophagus, small intestine, and the large intestine(colon).Gastrointestinal bleeding can result from regular use of some groups of medication, such as NSAIDs (e.g. aspirin), as well as antiplatelets such as clopidogrel and anticoagulants such as warfarin; however, these are required in some patients, especially those with states causing a tendency to form blood clots. Colon cancer is another potential cause gastrointestinal bleeding, thus iron deficiency anemia. Typically colon cancer occurs in older individuals[28] In addition, some bleeding disorders can cause gastrointestinal bleeding.[27] Two examples of bleeding disorders are von Willebrand disease and polycythemia vera.[27]

Diet In many countries, wheat flour is fortified with iron.[29]

The body normally gets the iron it requires from foods. If a person consumes too little iron, or iron that is poorly absorbed (non-heme iron), they can become iron deficient over time. Examples of iron-rich foods include meat, eggs, leafy green vegetables and iron-fortified foods. For proper growth and development, infants and children need iron from their diet.[30] For children, a high intake of cow’s milk is associated with an increased risk of iron-deficiency anemia.[31] Other risk factors for iron-deficiency anemia include low meat intake and low intake of iron-fortified products.[31]

The National Academy of Medicine updated Estimated Average Requirements (EARs) and Recommended Dietary Allowances (RDAs) in 2001. The current EAR for iron for women ages 14–18 is 7.9 mg/day, 8.1 for ages 19–50 and 5.0 thereafter (post menopause). For men the EAR is 6.0 mg/day for ages 19 and up. The RDA is 15.0 mg/day for women ages 15–18, 18.0 for 19–50 and 8.0 thereafter. For men, 8.0 mg/day for ages 19 and up. (RDAs are higher than EARs so as to identify amounts that will cover people with higher than average requirements.) The RDA for pregnancy is 27 mg/day, and for lactation, 9 mg/day. For children ages 1–3 years the RDA is 7 mg/day, 10 for ages 4–8 and 8 for ages 9–13.[32] The European Food Safety Authority (EFSA) refers to the collective set of information as Dietary Reference Values, with Population Reference Intakes (PRI) instead of RDAs, and Average Requirements instead of EARs. For women the PRI is 13 mg/day ages 15–17 years, 16 mg/day for women ages 18 and up who are premenopausal and 11 mg/day postmenopausal. For pregnancy and lactation, 16 mg/day. For men the PRI is 11 mg/day ages 15 and older. For children ages 1 to 14 the PRI increases from 7 to 11 mg/day. The PRIs are higher than the US RDAs, with the exception of pregnancy.[33]

Iron malabsorption

Iron from food is absorbed into the bloodstream in the small intestine, primarily in the duodenum.[34] Iron malabsorption is a less common cause of iron-deficiency anemia, but many gastrointestinal disorders can reduce the body's ability to absorb iron.[35] There are different mechanisms that may be present.

In celiac disease, abnormal changes in the structure of the duodenum can decrease iron absorption.[36] Abnormalities or surgical removal of the stomach can also lead to malabsorption by altering the acidic environment needed for iron to be converted into its absorbable form.[35] If there is insufficient production of hydrochloric acid in the stomach, hypochlorhydria/achlorhydria can occur (often due to chronic H. pylori infections or long-term proton pump inhibitor therapy), inhibiting the conversion of ferric iron to the absorbable ferrous iron.[36]

Pregnant women

Without iron supplementation, iron-deficiency anemia occurs in many pregnant women because their iron stores need to serve their own increased blood volume, as well as be a source of hemoglobin for the growing baby and for placental development.[30] Other less common causes are intravascular hemolysis and hemoglobinuria. Iron deficiency in pregnancy appears to cause long-term and irreversible cognitive problems in the baby.[37]


Babies are at increased risk of developing iron deficiency anemia due to their rapid growth.[24] Their need for iron is greater than they are getting in their diet.[24] Babies are born with iron stores; however, these iron stores typically run out by 4-6 months of age. In addition, infants who are given cow's milk too early can develop anemia due to gastrointestinal blood loss.[24]

Children who are at risk for iron-deficiency anemia include:[38]

  • Preterm infants
  • Low birth weight infants
  • Infants fed with cow milk under 12 months of age
  • Breastfed infants who have not received iron supplementation after age 6 months, or those receiving non-iron-fortified formulas
  • Children between the ages of 1 to 5 years old who receive more than 24 ounces (700 mL) of cow milk per day
  • Children with low socioeconomic status
  • Children with special health care needs
  • Children of Hispanic ethnicity [39]
  • Children who are overweight [39]
Blood donation

Frequent blood donors are also at risk for developing iron deficiency anemia.[40] When whole blood is donated, approximately 200mg of iron is also lost from the body.[41] The blood bank screens people for anemia before drawing blood for donation. If the patient has anemia, blood is not drawn.[41] Less iron is lost if the person is donating platelets or white blood cells.[41]


Anemia can result from significant iron deficiency.[35] When the body has sufficient iron to meet its needs (functional iron), the remainder is stored for later use in cells, mostly in the bone marrow and liver.[35] These stores are called ferritin complexes and are part of the human (and other animals) iron metabolism systems. Men store about 3.5g of iron in their body, and women store about 2.5g.[14]

Iron is a mineral that is important in the formation of red blood cells in the body, particularly as a critical component of hemoglobin.[22] About 70% of the iron found in the body is bound to hemoglobin.[14] Iron is primarily absorbed in the small intestine, in particular the duodenum and jejunum. Certain factors increase or decrease absorption of iron. For example, taking Vitamin C with a source of iron is known to increase absorption. Some medications such as tetracyclines and antacids can decrease absorption of iron.[14] After being absorbed in the small intestine, iron travels through blood, bound to transferrin, and eventually ends up in the bone marrow, where it is involved in red blood cell formation.[22] When red blood cells are degraded, the iron is recycled by the body and stored.[22]

When the amount of iron needed by the body exceeds the amount of iron that is readily available, the body can use iron stores (ferritin) for a period of time, and red blood cell formation continues normally.[35] However, as these stores continue to be used, iron is eventually depleted to the point that red blood cell formation is abnormal.[35] Ultimately, anemia ensues, which by definition is a hemoglobin lab value below normal limits.[35][3]

Diagnosis Blood smear of a person with iron-deficiency anemia at 40X enhancement

Conventionally, a definitive diagnosis requires a demonstration of depleted body iron stores obtained by bone marrow aspiration, with the marrow stained for iron.[42][43] However, with the availability of reliable blood tests that can be more readily collected for iron-deficiency anemia diagnosis, a bone marrow aspiration is usually not obtained.[44] Furthermore, a study published April 2009 questions the value of stainable bone marrow iron following parenteral iron therapy.[45] Once iron deficiency anemia is confirmed, gastrointestinal blood loss is presumed to be the cause until proven otherwise since it can be caused by an otherwise asymptomatic colon cancer. Initial evaluation must include esophagogastroduodenoscopy and colonoscopy to evaluate for cancer or bleeding of the gastrointestinal tract.

A thorough medical history is important to the diagnosis of iron-deficiency anemia. The history can help to differentiate common causes of the condition such as a menstruation in woman or the presence of blood in the stool.[46] A travel history to areas in which hookworms and whipworms are endemic may also be helpful in guiding certain stool tests for parasites or their eggs.[47] Although symptoms can play a role in identifying iron-deficiency anemia, they are often vague, which may limit their contribution to determining the diagnosis.

Blood tests Change in lab values in iron deficiency anemia Change Parameter ↓ ferritin, hemoglobin, MCV, MCH ↑ TIBC, transferrin, RDW, FEP

Anemia is often discovered by routine blood tests. A sufficiently low hemoglobin (Hb) by definition makes the diagnosis of anemia, and a low hematocrit value is also characteristic of anemia. Further studies will be undertaken to determine the anemia's cause. If the anemia is due to iron deficiency, one of the first abnormal values to be noted on a CBC, as the body's iron stores begin to be depleted, will be a high red blood cell distribution width (RDW), reflecting an increased variability in the size of red blood cells (RBCs).[22][16]

A low mean corpuscular volume (MCV) also appears during the course of body iron depletion. It indicates a high number of abnormally small red blood cells. A low MCV, a low mean corpuscular hemoglobin or mean corpuscular hemoglobin concentration (MCH), and the corresponding appearance of RBCs on visual examination of a peripheral blood smear narrows the problem to a microcytic anemia (literally, a small red blood cell anemia).[16]

The blood smear of a person with iron-deficiency anemia shows many hypochromic (pale, relatively colorless) and small RBCs, and may also show poikilocytosis (variation in shape) and anisocytosis (variation in size).[16][44] Target cells may also be seen. With more severe iron-deficiency anemia, the peripheral blood smear may show hypochromic, pencil-shaped cells and, occasionally, small numbers of nucleated red blood cells.[48] The platelet count may be slightly above the high limit of normal in iron-deficiency anemia (termed a mild thrombocytosis), but severe cases can present with thrombocytopenia (low platelet count).[49]

Iron-deficiency anemia is confirmed by tests that include serum ferritin, serum iron level, serum transferrin, and total iron binding capacity (TIBC). A low serum ferritin is most commonly found. However, serum ferritin can be elevated by any type of chronic inflammation and thus is not consistently decreased in iron-deficiency anemia.[22] Serum iron levels may be measured, but serum iron concentration is not as reliable as the measurement of both serum iron and serum iron-binding protein levels (TIBC).[18] The ratio of serum iron to TIBC (called iron saturation or transferrin saturation index or percent) is a value with defined parameters that can help to confirm the diagnosis of iron-deficiency anemia; however, other conditions must also be considered, including other types of anemia.[18]

Another finding that can be used is the level of free erythrocyte protoporphyrin (FEP).[50] During haemoglobin synthesis, trace amounts of zinc will be incorporated into protoporphyrin in the place of iron which is lacking. We can separate the protoporphyrin from its zinc moeity and measure it, known as the FEP, providing an indirect measurement of the zinc-protoporphyrin complex. The level of FEP is expressed in either μg/dl of whole blood or μg/dl of RBC. An iron insufficiency in the bone marrow can be detected very early by a rise in FEP.

Further testing may be necessary to differentiate iron-deficiency anemia from other disorders, such as thalassemia minor.[51] It is very important not to treat people with thalassemia with an iron supplement, as this can lead to hemochromatosis. A hemoglobin electrophoresis provides useful evidence for distinguishing these two conditions, along with iron studies.[18][52]


It is unclear if screening pregnant women for iron-deficiency anemia during pregnancy improves outcomes in the United States.[53] The same holds true for screening children who are 6 to 24 months old.[54] Even so, screening is a Level B recommendation suggested by the US Preventative Services Task Force in pregnant women without symptoms and in infants considered high risk. Screening is done with either a hemoglobin or hematocrit lab test.[39]

Treatment See also: Iron deficiency and Lucky iron fish Ascorbic acid

Treatment should take into account the cause and severity of the condition.[5] If the iron-deficiency anemia is a result of blood loss or another underlying cause, treatment is geared toward addressing the underlying cause.[5] Most cases of iron deficiency anemia are treated with oral iron supplements.[55] In severe acute cases, treatment measures are taken for immediate management in the interim, such as blood transfusions or intravenous iron.[5]

For less severe cases, treatment of iron-deficiency anemia includes dietary changes to incorporate iron-rich foods into regular oral intake and oral iron supplementation.[5] Foods rich in ascorbic acid (vitamin C) can also be beneficial, since ascorbic acid enhances iron absorption.[5] Oral iron supplements are available in multiple forms. Some are in the form of pills and some are drops for children.[5] Most forms of oral iron replacement therapy are absorbed well by the small intestine; however, there are certain preparations of iron supplements that are designed for longer release in the small intestine than other preparations.[55] Oral iron supplements are best taken up by the body on an empty stomach because food can decrease the amount of iron absorbed from the small intestine.[55] The dosing of oral iron replacement therapy is as much as 200 mg per day. This is generally spread out as 3-4 pills taken throughout the day.[55] The various forms of treatment are not without possible adverse side effects. Iron supplementation by mouth commonly causes negative gastrointestinal effects, including constipation.[56] Constipation is reported by 15-20% of patients taking oral iron therapy.[55] Preparations of iron therapy that take longer to be absorbed by the small intestine (extended release iron therapy) are less likely to cause constipation.[55] It can take six months to one year to get blood levels of iron up to a normal range and provide the body with iron stores.[55]

As iron-deficiency anemia becomes more severe, if the anemia does not respond to oral treatments, or if the treated person does not tolerate oral iron supplementation, then other measures may become necessary.[5][56] Two options are intravenous iron injections and blood transfusion.[55] Intravenous can be for people who do not tolerate oral iron or who require iron on a long term basis.[55] For example, people receiving dialysis treatment who are also getting erythropoietin or another erythropoiesis-stimulating agent are given parenteral iron, which helps the body respond to the erythropoietin agents to produce red blood cells.[56][57]

Intravenous iron can induce an allergic response that can be as serious as anaphylaxis, although different formulations have decreased the likelihood of this adverse effect.[56] In certain cases intravenous iron is both safer and more effective than the oral route.[58] For patients with severe anemia such as from blood loss, or who have severe symptoms such as cardiovascular instability, a blood transfusion may be considered.[55]

Epidemiology Deaths due to iron-deficiency anaemia per million persons in 2012 .mw-parser-output .refbegin{font-size:90%;margin-bottom:0.5em}.mw-parser-output .refbegin-hanging-indents>ul{list-style-type:none;margin-left:0}.mw-parser-output .refbegin-hanging-indents>ul>li,.mw-parser-output .refbegin-hanging-indents>dl>dd{margin-left:0;padding-left:3.2em;text-indent:-3.2em;list-style:none}.mw-parser-output .refbegin-100{font-size:100%}  no data  0  1  2-3  4-5  6-8  9-12  13-19  20-30  31-74  75-381 Disability-adjusted life year for iron-deficiency anemia per 100,000 inhabitants in 2004.[59]   no data   less than 50   50-100   100-150   150-200   200-250   250-300   300-350   350-400   400-450   450-500   500-1000   more than 1000

A moderate degree of iron-deficiency anemia affects approximately 610 million people worldwide or 8.8% of the population.[60] It is slightly more common in females (9.9%) than males (7.8%).[60] Up to 15% of children ages 1-3 years have iron deficiency anemia.[39] Mild iron deficiency anemia affects another 375 million.[60]

The prevalence of iron deficiency as a cause of anemia varies among countries; in the groups in which anemia is most common, including young children and a subset of non-pregnant women, iron deficiency accounts for a fraction of anemia cases in these groups (25% and 37%, respectively).[61] Iron deficiency is common in pregnant women.[62]

Within the United States, iron-deficiency anemia affects about 2% of adult males, 10.5% of Caucasian women, and 20% of African-American and Mexican-American women.[63]

A map provides a country-by-country listing of what nutrients are fortified into specified foods. Some of the Sub-Saharan countries shown in the deaths from iron-deficiency anemia map from 2012 are as of 2018 fortifying foods with iron.[29]

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External links ClassificationD
  • ICD-10: D50
  • ICD-9-CM: 280
  • MeSH: D018798
  • DiseasesDB: 6947
External resources
  • MedlinePlus: 000584
  • eMedicine: med/1188
  • The Importance of Iron – From IronTherapy.Org
  • Interactive material on Iron Metabolism – From
  • Establishing the cause of anemia – From
  • Handout: Iron Deficiency Anemia – From the National Anemia Action Council
  • NPS News 70: Iron deficiency anaemia: NPS – Better choices, Better health – From the National Prescribing Service
  • v
  • t
  • e
Diseases of red blood cells (D50–69,74, 280–287)↑Polycythemia
  • Polycythemia vera
  • Micro-: Iron-deficiency anemia
    • Plummer–Vinson syndrome
  • Macro-: Megaloblastic anemia
    • Pernicious anemia
(mostly normo-)Hereditary
  • enzymopathy: G6PD
  • glycolysis
    • PK
    • TI
    • HK
  • hemoglobinopathy: Thalassemia
    • alpha
    • beta
    • delta
  • Sickle-cell disease/trait
  • HPFH
  • membrane: Hereditary spherocytosis
    • Minkowski–Chauffard syndrome
  • Hereditary elliptocytosis
    • Southeast Asian ovalocytosis
  • Hereditary stomatocytosis
  • Autoimmune
    • WAHA
    • CAD
    • PCH
  • membrane
    • PNH
  • MAHA
  • TM
    • HUS
  • Drug-induced autoimmune
  • Drug-induced nonautoimmune
  • Hemolytic disease of the newborn
(mostly normo-)
  • Hereditary: Fanconi anemia
  • Diamond–Blackfan anemia
  • Acquired: PRCA
  • Sideroblastic anemia
  • Myelophthisic
Blood tests
  • MCV
    • Normocytic
    • Microcytic
    • Macrocytic
  • MCHC
    • Normochromic
    • Hypochromic
  • Methemoglobinemia
  • Sulfhemoglobinemia
  • Reticulocytopenia

Organic Dried Goji Berries by Alovitox | Raw, Vegan, Gluten Free Super Snack | High in Plant Based Protein, Dietary Fiber, Vitamin A & Iron | Extra Large Berries for Eating, Trail Mixes or Smoothies
Organic Dried Goji Berries by Alovitox | Raw, Vegan, Gluten Free Super Snack | High in Plant Based Protein, Dietary Fiber, Vitamin A & Iron | Extra Large Berries for Eating, Trail Mixes or Smoothies
Several traditional and folk healing methods around the world have recognized the many medicinal properties attributed to this sweet and tart berry. Goldenberries contain many vitamins and minerals;however, the real value of this fruit is the antioxidant concentration. Polyphenols and carotenoids are powerful elements of human health, and golden berries contain significant amounts. Specifically, Goldenberries are a good source of Vitamin A, with 25% of recommended daily value per serving. In addition, with 10% of recommended daily value per serving, Goldenberries are also a good source of Potassium. Goldenberries are a healthy food. The statements herein have not been reviewed by the FDA. Alovitox Naturals products are not intended to treat, diagnose, cure or prevent any disease.

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Snap and Giggle Raw Organic Cacao Nibs, Sugar Free Chocolate Chips, Excellent for Keto, Paleo, and Vegan Snacks, Natural Flavor, High in Fiber, Magnesium, and Iron, 226 Grams
Snap and Giggle Raw Organic Cacao Nibs, Sugar Free Chocolate Chips, Excellent for Keto, Paleo, and Vegan Snacks, Natural Flavor, High in Fiber, Magnesium, and Iron, 226 Grams
Cacao nibs are chocolate in its purest form. They are a part of the cacao bean, that has been harvested, fermented, dried, cleaned, roasted, cracked, and winnowed, or removed from the shell. The texture of cacao nibs is said to be similar to that of roasted coffee beans, with a slightly bitter and nutty flavor profile. Cacao nibs are the less processed version of chocolate with no unhealthy additives, and all the preserved nutrients of the natural cacao bean.

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Honey Maid Cinnamon Graham Crackers, 14.4 Ounce
Honey Maid Cinnamon Graham Crackers, 14.4 Ounce
For nearly 90 years, Honey Maid has been a wholesome part of snack time. Honey Maid makes s’mores fun for the whole family or simply serve as inspiration to create a delicious snack.

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ProtiDIET Delicious Protein Bar | Nutritious Low Fat & Carb Snack With High Vitamins & Minerals | | Healthy & Energizing Small Meal | Assists In Weight Loss (Banana Bread)
ProtiDIET Delicious Protein Bar | Nutritious Low Fat & Carb Snack With High Vitamins & Minerals | | Healthy & Energizing Small Meal | Assists In Weight Loss (Banana Bread)
Wake up to the rich and savory taste of freshly baked banana bread enhanced with a touch of chocolate goodness. ProtiDiet's Banana Bread Breakfast Bar helps you start your day right with 15 grams of energizing protein, 5 grams of healthy fiber and 10% of your Daily Value of Iron. Each delicious breakfast protein bar delivers all this nutrition and great taste for just 150 calories. Breakfast is served!

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Al Ajwa Dates a Superfood, High Fiber, Heart Healthy Snacks - Natural Dried Fruit Supplement and Energy Booster - Ready to Eat Food, 800g
Al Ajwa Dates a Superfood, High Fiber, Heart Healthy Snacks - Natural Dried Fruit Supplement and Energy Booster - Ready to Eat Food, 800g
NATURALLY NUTRITIOUS: Al Ajwa Dates are rich in healthy fiber, minerals and vitamins. Dates contain soluble fiber (it draws water into the digestive tract) and can be a constipation relief. Dates are a source of iron and can help prevent anemia. They contain magnesium, manganese and selenium. NATURAL ENERGY BOOSTER: Now you can skip energy bars and energy drinks that contain processed sugar like high fructose corn syrup. Eat dates instead. Dates are filled with natural sugars and can be consumed in small quantities due to its tiny size. This is the secret weight loss food you need to have a "Live strong, Healthy diet". STAY HEALTHY WHEN TRAVELLING: Adding dates to oatmeal or muesli gives you a natural, ready to eat food when you are on the move or too busy. THEY TASTE GREAT: This is a tiny superfood that tastes great on its own.

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Food to Live Pepitas/Pumpkin Seeds (Raw, No Shell, Kosher) (2 Pounds)
Food to Live Pepitas/Pumpkin Seeds (Raw, No Shell, Kosher) (2 Pounds)
Pepitas are high in protein, phosphorous and iron. Great in salads or just for snacking. No need to worry about what to do with the shells because there aren't any! Pepitas are extremely popular in Mexico where they are toasted and spiced with salt, chile or other flavors for a great snack. You cooks, and foodies also know that grinding them up provides the essential ingredient for pipian, a type of mole.

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Organic Green Split Peas by Food to Live (Non-GMO, Kosher, Raw, Dried, Great for Pea Soup, High in Protein and Fiber, Bulk, Product of Canada) — 20 Pounds
Organic Green Split Peas by Food to Live (Non-GMO, Kosher, Raw, Dried, Great for Pea Soup, High in Protein and Fiber, Bulk, Product of Canada) — 20 Pounds
One of the first food crops ever to be cultivated, probably originated in the Mediterranean region and Western Asia. High in fiber, protein and carbohydrates, iron, potassium, vitamin A, thiamine and riboflavin, peas provide excellent nutrition at a very low cost. They can be grown in dark or light. When grown completely in the dark, the sprouts have the look and taste of thin yellow asparagus. Health Benefits: Green peas are one of the most nutritious leguminous vegetables, rich in health benefiting phyto-nutrients, minerals, vitamins and anti-oxidants. Peas are relatively low in calories on comparison with beans, and cowpeas. 100 g of green peas provide only 81 calories, and no cholesterol. Nonetheless, the legumes are a good source of proteins, and soluble as well as insoluble fiber. Fresh pea pods are excellent source of folic acid. 100 g provides 65 µg or 16% of recommended daily levels of folates. Folates are B-complex vitamins required for DNA synthesis inside the cell. Well established research studies suggest that adequate folate rich foods in expectant mothers would help prevent neural tube defects in the newborn babies. Fresh green peas are very good in ascorbic acid (vitamin C). Contain 40 mg/100 g or 67% of daily requirement of vitamin C. Vitamin C is a powerful natural water-soluble anti-oxidant. Vegetables rich in this vitamin helps body develop resistance against infectious agents and scavenge harmful, pro-inflammatory free radicals from the body. Peas contain phytosterols especially ß-sitosterol. Studies suggest that vegetables like legumes, fruits and cereals rich in plant sterols help lower cholesterol levels in the body. Nutritional info: Vitamins A, B, C and E Calcium, Iron, Phosphorus Amino Acids Protein: 25%.

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Special K Nourish Cereal, Berries and Peaches with Probiotics, 15.5 oz
Special K Nourish Cereal, Berries and Peaches with Probiotics, 15.5 oz
Nourish your strength when you start each day with Special K Nourish Probiotics, Berries and Peaches—a deliciously crafted, wholesome cereal that makes your morning a little brighter with each irresistible spoonful. This filling cereal is a satisfying mix of crunchy multi-grain flakes, sweet yogurty pieces that contain probiotics and real peaches, blueberries, and raspberries to energize you for the day ahead. Just as nutritious as it is delicious, this breakfast cereal contains whole grain, fiber, and essential vitamins and minerals to help keep you going strong. Whether you enjoy a bowlful at breakfast time, add it to your yogurt for a snack at the office, or enjoy it straight from the box after your workout, Kellogg's Special K Nourish Berries and Peaches with Probiotics cereal lets you own your day with each delicious bite.

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NUTS U.S. - Organic Sun-Dried Apricots (3 LB.)
NUTS U.S. - Organic Sun-Dried Apricots (3 LB.)
If you are a health cautious individual who likes to stay active and enjoy healthylifestyle and all natural snacks then these Organic Dried Apricots are for YOU! These apricots have a distinctive flavor you will immediately notice! Our PREMIUM dried apricots are - Packed fresh - TASTY - and kept in SEALED bags for extra security and freshness - Bags are resealable so that you can enjoy them FRESH every time. Dried Apricots are highly nutritious, they are rich in fiber, antioxidants, and potassium. These nuts are great for snacking, making salads, or baking. Don't waste any more time and enjoy these delicious, dried berries that people have been enjoying for decades!

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Science in Sport Go Energy Bar | Apple & Blackcurrant Flavor - Carbohydrate Endurance Snack Bars - 20 Pack (40 Gram)
Science in Sport Go Energy Bar | Apple & Blackcurrant Flavor - Carbohydrate Endurance Snack Bars - 20 Pack (40 Gram)
SiS GO Energy bars are a nutritious, easily digestible and high carbohydrate based snack made from fruits and wholesome ingredients. The reduced size (compared to the larger GO Energy Bars) provides a practical and nutritious approach to provide carbohydrate during exercise. Proven by thousands of athletes in the world's toughest races, it meets their demanding energy needs.

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